TNF-alpha up or down? Winthrop must
answer your questions and make adequate disclosure.
contact 516-663-9582
dwhitfield@winthrop.org
See faustmanlab.org and pubmed.org faustman dl and pubmed.org ristori bcg
see below and post your questions comments
https://www.winthrop.org/departments/clinical/ctn/
https://www.winthrop.org/research/research-areas/Rheumatology-Allergy-Immunology.cfm
Rheumatoid Arthritis
Summary
Winthrop University Hospital, Clinical Trials Center is conducting a clinical research study to test the effectiveness of the study drug in reducing pain and maintaining movement in joints for participants who are at least 18 years old and diagnosed with active Rheumatoid Arthritis.Description
The purpose of this research study is to further examine an investigational medication for the treatment of Rheumatoid Arthritis. The study drug is FDA approved as a subcutaneous (under the skin) injection but when given as an IV infusion study drug is investigational (not approved by the FDA).Inclusion/exclusion
Eligible participants will receive study-related medical examinations, research medication and study-related laboratory tests at no cost, while they are participating in this research study. Compensation may be provided for time and travel. You may be able to participate if you are; ¥ at least 18 years old ¥ diagnosed with Rheumatoid Arthritis ¥ have had inadequate treatment results with other medicationsPrincipal Investigator
Steven Carsons, MDDepartment
Clinical Trials CenterOther Contact
for more information please call: 516-663-9582Result Filters
Cell Mol Life Sci. 2005 Aug;62(16):1850-62.
The therapeutic potential of tumor necrosis factor for autoimmune disease: a mechanistically based hypothesis.
Abstract
Excess
levels of tumor necrosis factor-alpha (TNF-alpha) have been associated
with certain autoimmune diseases. Under the rationale that elevated
TNF-alpha levels are deleterious, several anti-TNF-alpha therapies are
now available to block the action of TNF-alpha in patients with
autoimmune diseases with a chronic inflammatory component to the
destructive process. TNF-alpha antagonists have provided clinical
benefit to many patients, but their use also is accompanied by new or
aggravated forms of autoimmunity. Here we propose a mechanistically
based hypothesis for the adverse events observed with TNF-alpha
antagonists, and argue for the opposite therapeutic strategy: to boost
or restore TNF-alpha activity as a treatment for some forms of
autoimmunity. Activation defects in the transcription factor nuclear
factor kappaB leave autoreactive T cells sensitive to TNF-alpha-induced
apoptosis. Treatment with TNF-alpha, by destroying autoreactive T cells,
appears to be a highly targeted strategy to interrupt the pathogenesis
of type 1 diabetes, lupus and certain forms of autoimmunity.
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Front Immunol. 2013 Dec 23;4:478. eCollection 2013.
TNF Receptor 2 and Disease: Autoimmunity and Regenerative Medicine.
Faustman DL1, Davis M2.
Abstract
THE
REGULATORY CYTOKINE TUMOR NECROSIS FACTOR (TNF) EXERTS ITS EFFECTS
THROUGH TWO RECEPTORS: TNFR1 and TNFR2. Defects in TNFR2 signaling are
evident in a variety of autoimmune diseases. One new treatment strategy
for autoimmune disease is selective destruction of autoreactive T cells
by administration of TNF, TNF inducers, or TNFR2 agonism. A related
strategy is to rely on TNFR2 agonism to induce T-regulatory cells (Tregs)
that suppress cytotoxic T cells. Targeting TNFR2 as a treatment
strategy is likely superior to TNFR1 because of its more limited
cellular distribution on T cells, subsets of neurons, and a few other
cell types, whereas TNFR1 is expressed throughout the body. This review
focuses on TNFR2 expression, structure, and signaling; TNFR2 signaling
in autoimmune disease; treatment strategies targeting TNFR2 in
autoimmunity; and the potential for TNFR2 to facilitate end organ
regeneration.
KEYWORDS:
TNF, TNF receptor 2, autoimmune disease, regeneration, type 1 diabetes- PMID:
- 24391650
- [PubMed - as supplied by publisher]
- PMCID:
- PMC3870411
Result Filters
Sci Rep. 2013 Nov 6;3:3153. doi: 10.1038/srep03153.
Homogeneous expansion of human T-regulatory cells via tumor necrosis factor receptor 2.
Author information
- 1Immunobiology Laboratory, Massachusetts General Hospital and Harvard Medical School, Rm 3602, MGH-East, Bldg 149, 13th Street, Boston, MA 02129.
Abstract
T-regulatory
cells (T(regs)) are a rare lymphocyte subtype that shows promise for
treating infectious disease, allergy, graft-versus-host disease,
autoimmunity, and asthma. Clinical applications of T(regs) have not been
fully realized because standard methods of expansion ex vivo produce
heterogeneous progeny consisting of mixed populations of CD4 + T cells.
Heterogeneous progeny are risky for human clinical trials and face
significant regulatory hurdles. With the goal of producing homogeneous
T(regs), we developed a novel expansion protocol targeting tumor
necrosis factor receptors (TNFR) on T(regs). In in vitro studies, a
TNFR2 agonist was found superior to standard methods in proliferating
human T(regs) into a phenotypically homogeneous population consisting of
14 cell surface markers. The TNFR2 agonist-expanded T(regs) also were
functionally superior in suppressing a key T(reg) target cell, cytotoxic
T-lymphocytes. Targeting the TNFR2 receptor during ex vivo expansion is
a new means for producing homogeneous and potent human T(regs) for
clinical opportunities.
- PMID:
- 24193319
- [PubMed - in process]
- PMCID:
- PMC3818650
Ristori
G, Romano S, Cannoni S, Visconti A, Tinelli E, Mendozzi L, Cecconi P,
Lanzillo R, Quarantelli M, Buttinelli C, Gasperini C, Frontoni M,
Coarelli G, Caputo D, Bresciamorra V, Vanacore N, Pozzilli C, Salvetti
M.
Neurology. 2014 Jan 7;82(1):41-8. doi: 10.1212/01.wnl.0000438216.93319.ab. Epub 2013 Dec 4.
- PMID:
- 24306002
- [PubMed - indexed for MEDLINE]
2.
Paolillo A, Buzzi MG, Giugni E, Sabatini U, Bastianello S, Pozzilli C, Salvetti M, Ristori G.
J Neurol. 2003 Feb;250(2):247-8. No abstract available.
- PMID:
- 12622098
- [PubMed - indexed for MEDLINE]
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Help Support Our
Phase II Clinical Trial!
Phase II Clinical Trial!
We have raised $17.2 million for the Phase II
human clinical trial testing BCG in type 1 diabetes. Our total need:
$25.2 million. Your donation will directly support this research.
-------------
The Faustman Lab at Massachusetts General Hospital
Denise Faustman, MD, PhD, is Director of the Immunobiology
Laboratory at the Massachusetts General Hospital (MGH) and an Associate
Professor of Medicine at Harvard Medical School. Her current research
focuses on discovering and developing new treatments for type 1 diabetes
and other autoimmune diseases, including Crohn's disease, lupus,
scleroderma, rheumatoid arthritis, Sjögren's syndrome, and multiple
sclerosis. She is currently leading a human clinical trial program
testing the efficacy of the BCG vaccine for reversal of long-term type 1
diabetes. Positive results from the Phase I study were reported in 2012.Dr. Faustman's type 1 diabetes research has earned her notable awards such as the Oprah Achievement Award for “Top Health Breakthrough by a Female Scientist” (2005), the "Women in Science Award" from the American Medical Women’s Association and Wyeth Pharmaceutical Company for her contributions to autoimmune disease research (2006), and the Goldman Philanthropic Partnerships/Partnership for Cures “George and Judith Goldman Angel Award” for research to find an effective treatment for type 1 diabetes (2011). Her previous research accomplishments include the first scientific description of modifying donor tissue antigens to change their foreignness. This achievement earned her the prestigious National Institutes of Health and National Library of Medicine “Changing the Face of Medicine” Award (2003) as one of 300 American physicians (one of 35 in research) honored for seminal scientific achievements in the United States.
Dr. Faustman earned her MD and PhD from Washington University School of Medicine in St. Louis, Missouri, and completed her internship, residency, and fellowships in Internal Medicine and Endocrinology at the Massachusetts General Hospital in Boston, Massachusetts.
Result Filters
Ann Neurol. 1995 Aug;38(2):147-54.
Reduced expression of peptide-loaded HLA class I molecules on multiple sclerosis lymphocytes.
Abstract
Lymphocytes from patients with HLA class II-linked autoimmune diseases such as type I diabetes, systemic lupus erythematosus, rheumatoid
arthritis, and Graves' have recently been shown to have a decrease in
the expression of self-peptide-filled HLA class I antigens on the
surface of peripheral lymphocytes. The human demyelinating diseases of
multiple sclerosis in some cases are also associated with the presence
of certain HLA class II genes, which may in turn be linked to genes in
the class II region that control class I expression. Hence, we studied
fresh peripheral blood mononuclear cells (PBMCs) and newly produced
Epstein-Barr virus (EBV)-transformed cell lines from multiple sclerosis
patients for the class I defect. Unseparated PBMCs, as well as T cells, B
cells, and macrophages from multiple sclerosis patients had a decrease
in the amount of conformationally correct peptide-filled HLA class I
molecules on the cell surface compared with matched controls detectable
by flow cytometry. To demonstrate the independence of this defect from
exogenous serum factors, newly produced EBV-transformed cell lines from B
cells of patients with multiple sclerosis maintained the defect. In
addition, DR2 +/+, +/-, and -/- EBV-transformed B cells from these
patients similarly demonstrated the self-antigen presentation defect.
Analysis of a set of discordant multiple sclerosis twins revealed the
class I defect was exclusively found on the affected twin lymphocytes,
suggesting a role of this class I complex in disease expression. These
data indicate that multiple sclerosis patients have abnormal
presentation of self-antigens.(ABSTRACT TRUNCATED AT 250 WORDS)
Comment in
- PMID:
- 7654061
- [PubMed - indexed for MEDLINE]
J Clin Invest. 1993 May;91(5):2301-7.Defective major histocompatibility complex class I expression on lymphoid cells in autoimmunity.
Abstract
Lymphocytes from patients with insulin-dependent diabetes mellitus (IDDM), a chronic autoimmune disease, have recently been shown to have decreased surface expression of MHC class I antigens. Since IDDM and other autoimmune diseases share a strong genetic association with MHC class II genes, which may in turn be linked to genes that affect MHC class I expression, we studied other autoimmune diseases to determine whether MHC class I expression is abnormal. Fresh PBLs were isolated from patients with IDDM, Hashimoto's thyroiditis, Graves' disease, systemic lupus erythematosis, rheumatoid arthritis, and Sjogren's syndrome. Nondiabetic and non-insulin-dependent diabetes mellitus patients served as controls. MHC class I expression was measured with a conformationally dependent monoclonal antibody, W6/32. Freshly prepared PBLs from the autoimmune diseases studied and the corresponding fresh EBV-transformed B cell lines had decreased MHC class I expression compared with PBLs from normal volunteers and non-insulin-dependent (nonautoimmune) diabetic patients. Only 3 of more than 180 donors without IDDM or other clinically recognized autoimmune disease had persistently decreased MHC class I expression; one patient was treated with immunosuppressive drugs, and subsequent screening of the other two patients revealed high titers of autoantibodies, revealing clinically occult autoimmunity. Patients with nonautoimmune inflammation (osteomyelitis or tuberculosis) had normal MHC class I expression. Autoimmune diseases are characterized by decreased expression of MHC class I on lymphocytes. MHC class I expression may be necessary for self-tolerance, and abnormalities in such expression may lead to autoimmunity.
J Clin Immunol. 1993 Jan;13(1):1-7.Mechanisms of autoimmunity in type I diabetes.
Abstract
The work presented in this review suggests that in human and murine type I diabetes, defective MHC class I expression on APC is linked to autoimmunity. The defect in self-antigen presentation is present on prediabetic and diabetic APC, and this presumably delivers abnormal or lack of signals to T cells to allow self tolerance. Since most autoimmune diseases have strong genetic linkage to MHC class II region, our recent results additionally demonstrating low MHC class I expression on lymphoid cells in a diversity of autoimmune diseases (hypothyroidism, rheumatoid arthritis, lupus, etc.) suggest that this pathway of abnormal class I presentation of self epitopes may be important for tolerance to many tissue-specific antigens (40). Certainly, the unanswered genetic questions will address the role of the specific genes controlling self-antigen presentation through MHC class I followed by T-cell education to self.
Don't get deceived, defrauded, scammed, and/or receive incomplete disclosure from Winthrop whether affirmatively required or not.
Thanks for your support!
I understand your interest in the BCG vaccine, however until we carry out our planned studies, we do not have sufficient scientific evidence of the effect.
The BCG vaccine is available at for all persons travelling to areas where tubersulosis is endemic. Please ask your general practitioner to guide you to where you could have the vaccine.
Best wishes,
Lone Graff Stensballe
Sendt: 7. september 2012 01:53
Til: Lone Graff Stensballe
Emne: FW: Tuberculosis vaccine - a new remedy for allergies and asthma in children?
lone.graff.stensballe@rh.regionh.dk:
I would like to be vaccinated with BCG and obtain access to the same as I am a Type 1 diabetic and believe that BCG will treat and/or cure my diabetes and perhaps my other autoimmune diseases eg plaque psoriasis. My brother also suffers from Type 1 diabetes as did my fraternal uncle. My first cousin has Sjorgrens.
I would assume you are familiar with the work of Dr. Denise L Faustman. See eg faustmanlab.org and pubmed.org faustman dl.
Do you have any colleagues in the US that would vaccinate me with BCG and administer same. I would like to make a You Tube video of the usefulness of BCG for Type 1 diabetes.
Sincerely yours,
Date: Thu, 6 Sep 2012 17:37:59 -0400
From: Delthia.Ricks@newsday.com
Subject: Fwd: Tuberculosis vaccine - a new remedy for allergies and asthma in children?
>>> Rigshospitalet <news@meltwaterpress.com> 9/3/2012 9:27 AM >>>
Press release
3rd of September 2012
Tuberculosis vaccine - a new remedy for allergies and asthma in children?
M Can a vaccine against tuberculosis help combat asthma and eczema in Danish children early in life? This will now be examined in a comprehensive Danish research study.
From September 2012, thousands of Danish pregnant women will receive an invitation to allow their newborns to take part in a sensational trial.
The tuberculosis vaccine was removed from the vaccine program in Denmark during the 1980s, however new research indicates that the vaccine can improve the health of children.
Research carried out in developing countries shows that the health of infants who have been given the tuberculosis vaccine (BCG/Calmette) at birth is improved and the babies have a better survival rate than those who have not been given the vaccine. The vaccine also seems to have a preventive effect against asthma and atopic dermatitis.
Results are so striking that they cannot be explained by the fact that the children did not catch tuberculosis. Therefore, researchers assess the vaccine to have a general positive effect on the immune system, which means that children are less sick, and have less atopic dermatitis, asthma and allergies.
Whether this positive effect also can benefit Danish children will now be examined in a large Danish research project headed by Lone Graff Stensballe, Paediatrician from the Department of Paediatrics and Adolescent Medicine at Rigshospitalet.
The research project will run for three years, starting in September 2012, where 4,300 infants and their parents will be followed through interviews, examinations, and, for 300 of the children, blood tests as well. The project will comprise five PhD courses and a research collaboration with obstetricians, paediatricians, midwives, nurses and laboratory technicians from the three hospitals taking part in the project.
“We are very excited about this unique opportunity to improve the health of Danish children early in life,” says Lone Graff Stensballe. “Unfortunately, we have seen large increases in admissions, consumption of medicines, asthma, eczema and allergies among Danish children. We hope to curb these increases with the new research project.”
The research project will be carried out at Rigshospitalet in collaboration with Hvidovre Hospital, Kolding Sygehus Lillebælt and the new Centre for Vitamins and Vaccines at SSI (Statens Serum Institut).
For further information and interviews, please contact:
Lone Graff Stensballe
Head of Research
Paediatrician, Department of Paediatrics and Adolescent Medicine, Rigshospitalet, Denmark
Telephone: +45 6022 8092 E-mail: lone.graff.stensballe@rh.regionh.dk
Rigshospitalet - a part of Copenhagen University Hospital
Rigshospitalet – a part of Copenhagen University Hospital – is Denmark'sleading hospital for patients needing highly specialized treatment. Rigshospitalet serves all of Denmark, Greenland and the Faroe Islands within almost all specialties and sub-specialties of medicine and surgery.
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Decide for yourself whether it is in your interest to be vaccinated for TB with BCG a tried, true and inexpensive vaccine that has been used all over the world since 1921. Healthcare need not be expenisve and/or dangerous.
Plaque psoriasis is very easily visually documented
Doctors Without Borders starts new fundraising drive vaccinating Mineola residents with BCG to lessen their chance of contracting multidrug resident TB with the added benefit of reducing the impact of concommittant autoimmune diseases. Doctors Without Borders reads the work of Dr. Denise L
Faustman and finds it persuasive while Winthrop could not care less about simple, inexpensive, and useful treatments.
Take vaccines and TB for example: Vaccines have long been part of the services MSF offers, particularly in countries where vaccine coverage is low. As part of basic health care programs in these places, teams provide children with a slate of vaccines currently recommended by the World Health Organization: DTP (diphtheria, tetanus, pertussis), hepatitis B, Haemophilius influenzae type b (Hib), BCG (against tuberculosis), measles, polio, and, increasingly, pneumococcal conjugate vaccine.
Speech to Universities Allied for Essential Medicines
"In
MSF, people come and go. Some do one field assignment. Others – like me
- are involved with the organization for much longer. Regardless how
long we've worked for this organization, our experience stays in us
forever and changes our vision of humanity."
-Sophie Delaunay, MSF-USA Executive Director
-Sophie Delaunay, MSF-USA Executive Director
November 16, 2013
Then some patients came to us with a Chinese drug smuggled into Thailand through Burma. This drug was based on artemisinin derivatives. This family of new drugs was poorly known outside China. In fact, during the Vietnam War, for both the US and the Vietnamese forces, malaria was a real burden, keeping large numbers of soldiers out of combat. The Vietnamese asked their Chinese ally to provide them with better treatments. Chinese academics first screened the plants used in their traditional medicine. This is how the artemisinin derivatives were re-discovered. Meanwhile, Western laboratories, especially the US military research institutions, identified several new molecules, among them mefloquine and halofantrine.
The Vietnam War was over before this new generation of drugs had any impact on the treatment of military forces. Nonetheless, the two US drugs were developed and we used them in our programs. Later on, the parasite, Plasmodium falciparum, rapidly developed resistance against mefloquine and halofantrine. This is where the story starts for us and when we turned to the Chinese version.
With the Chinese drugs, three main difficulties immediately emerged: first of all, these drugs had not been studied according to international scientific standards; secondly, they were not economically attractive for big PHARMA to invest in because malaria was a disease of the poor, and also because the initial products were not patentable by Western pharmaceutical companies under the Chinese law at the time. And thirdly, politically, even psychologically, it was difficult for Western institutions to recognize that Chinese medical institutions had won the medical battle and found an alternative to outdated malaria drugs.
The first step out of this deadlock was to carry out clinical trials using alternative treatments. This was absolutely necessary if we were to have a chance at developing an effective treatment for one of the world’s most common and deadly diseases before it became completely untreatable. Looking at epidemiological data, it was obvious that there was a crucial need for a new treatment for malaria in other parts of the world, too. At the end of the nineties, the situation was rapidly worsening in sub-Saharan Africa. So between 1996 and 2004, MSF enrolled 12,000 patients in 43 clinical trials in 18 countries. Our efficacy studies were conducted according to scientific standards and published in peer-reviewed journals, and provided evidence on the efficacy of most drug regimens containing artesunate. We also supported the founding of the Drugs for Neglected Diseases initiative (DNDi) in 2003, whose mission was to fast track the development of drugs for neglected diseases. In addition to assisting in the development of new regimens to treat sleeping sickness, visceral leishmaniasis, and Chagas disease, DNDi helped create ASAQ and ASMQ, two single-dose combination treatments for malaria that include artemisinin and that are in widespread use in Africa and Asia today.
Artemisinin-based treatments are now part of WHO recommended protocols and have become a reference therapeutic option for malaria everywhere in the world. But as we speak, our teams in Cambodia are seeing the first signs of resistance to this treatment. We are already certain that this resistance will continue to spread and we can even anticipate which route it’ll take. But there is no single alternative treatment in sight.
The reason I shared this story on malaria with you is that in my view it is emblematic of a number of challenges that we still face today in our medical interventions: first, the lack of needs-driven innovation and research, especially when patients are poor and neglected; second, the persistence of numerous barriers (mostly economic) to access; and third, the need to fundamentally change the system in the long-run to promote innovation and access.
In my talk today, I will focus primarily on TB, vaccines, and hepatitis C, three areas where innovation and access gaps in the response from the global health system are currently causing tremendous suffering among the patients we serve. These are going to be three key priorities for MSF in the coming years. The fight for access to medicines by MSF always originates from a very concrete and practical problem faced by patients. It is not a moral or intellectual campaign that we embrace simply because we think it is the right cause.
Take vaccines and TB for example: Vaccines have long been part of the services MSF offers, particularly in countries where vaccine coverage is low. As part of basic health care programs in these places, teams provide children with a slate of vaccines currently recommended by the World Health Organization: DTP (diphtheria, tetanus, pertussis), hepatitis B, Haemophilius influenzae type b (Hib), BCG (against tuberculosis), measles, polio, and, increasingly, pneumococcal conjugate vaccine.
According to WHO estimates, globally in 2012 more than 22 million children did not benefit from basic vaccination. One big barrier to better coverage is that in rural areas of resource-limited settings, parents have to travel far to get their children vaccinated, simply because in many cases, it’s not possible to bring the vaccines to them. At present, these vaccines have storage and delivery requirements that can be very difficult to uphold in resource-limited settings. Imagine trying to reach children in a remote village in, say, India, or Chad, or Central African Republic. The roads are bad and prone to flooding during the rainy season. The vaccines themselves need to be kept at a certain temperature—between 2° to 8° Celsius, or roughly 35° to 46° Fahrenheit—lest they become ineffective, but there’s no electricity along much of the route. And most vaccines have to be administered with needles by trained health professionals, who are often in short supply in countries lagging in terms of development or afflicted by years of conflict. MSF teams must contend with all of these challenges, from the logistics of the cold chains, to the provision of trained medical staff. There is a profound need for vaccines that are better-adapted to the settings in which they will be used. If they could withstand higher temperatures, if they could be delivered through a mist or a patch, or if the course could be completed with fewer doses, it would be far more feasible to get the vaccines and health workers to the most decentralized health facilities, closer to where most people live. Rather than sticking with a one-size-fits-all approach—that is usually based on US and European models—vaccines R&D efforts should be much more global, responding to the needs of all children and the health systems that try to reach them.
Moving on to tuberculosis: After more than 4 decades of inaction, as we speak, two new promising drugs for TB are coming to market, but very little effort has been made by their producers to assess the drugs’ efficacy in new combination treatments or in sharing clinical safety data that would allow others to do this work. Effective TB treatment requires a robust combination of different classes of drugs to prevent development of resistance to individual drugs. So, even though the advent of new promising TB drugs is a milestone for us and for the 30,000 TB patients we treat in a year, these new drugs won’t actually do much to curb the drug-resistant TB epidemic until we know how to combine them and what kind of regimen is appropriate for treatment, and results from the necessary clinical trials are still many years away. Furthermore, producers have little incentive to make these new drugs affordable for all those who, from South Africa to Uzbekistan, are currently dying from drug-resistant TB and who desperately need access to new and better treatment regimens. So, as with malaria 20 years ago, big pharma lacks any incentive to perform the research necessary to meet patient needs. Companies are incentivized to work in silos to bring individual drugs to market, rather than to perform the collaborative research needed to develop and bring new regimens to market. We face a future where eventually treatment providers and NGOs will have to step up and conduct clinical trials themselves as we had to in malaria.
And that is why we need a fundamental change in the way in which medical research and development is conducted, and this is what MSF, UAEM, and many others are trying to enact through the WHO. As part of the implementation of the 2008 WHO Global Strategy and Plan of Action on Public Health, Innovation, and Intellectual Property and some of the recommendations included in the 2012 report by the Consultative Expert Working Group on Research and Development: Financing and Coordination (the CEWG Report), MSF has submitted two proposals for demonstration projects on innovative incentive and funding mechanisms for R&D: one for open-source, multiplex, affordable fever diagnostics and another for a new framework for TB regimen development.
Allow me to briefly explain our TB proposal, called the 3P Project (Push, Pull, Pool). We are proposing the creation of an open, collaborative framework for TB drug regimen development based on the sharing of data, the pooling of intellectual property, and the creation of incentives in the form of prizes and grants. Our proposal encourages multiple actors to enter the R&D process in order to accelerate development timelines, improve R&D effectiveness, and provide more equitable access to better medicines. The proposal de-links the costs of research and development from the prices of the end products created (known as “delinkage”) and embraces open, collaborative innovation models.
Your help in supporting our efforts to explain the CEWG process and the MSF proposals (as well as other proposals) to academics, researchers, universities, and many others would be extremely valuable. We need to broaden the discussion beyond the corridors of Geneva.
MSF has also launched an online TB manifesto advocating for better treatments that I encourage you all to sign and share broadly.
In other areas however, challenges of access and affordability are quite obvious. After more than 40 years with no new drugs, the newly FDA-approved TB treatment bedaquiline will most likely cost around 900 USD for a 6-month treatment course in least developed countries, and 3,000 USD for a six-month course in middle income countries. That’s for just one drug, but several drugs are needed for effective TB treatment. This means that new treatment combinations will still cost in excess of several thousand dollars per patient even in the poorest countries, as they do today , and potentially much more in countries classified as ‘middle-income,’ of which many have a high burden of TB, such as India, Ukraine and Uzbekistan.
At least 185 million people worldwide have been infected with hepatitis C virus (HCV), but access to treatment is and will continue to be a huge challenge. The current treatment peginterferon is expensive and not ideal in terms of medical outcomes, but some very promising new hepatitis C oral treatments are coming out of the pipeline very soon. The problem is that they are going to be completely unaffordable for many. Let me give you one example: sofubsuvir from Gilead is going to receive FDA and European regulatory approval very soon. According to the latest estimates, it could be priced in the range of 50-80,000 USD for a treatment course in wealthy countries. And there is yet no public indication on what Gilead will charge for its use in developing countries. We know that competition, if allowed, could be a game changer because the cost of manufacturing has been estimated to be around 200 USD per treatment.
Bringing prices of vaccines down is another crucial priority. In 2001, the cost to provide a child with basic vaccinations was $1.38. Today, it’s $38.80, an increase of more than 2,700 percent. Given the fact that the number of basic antigens recommended by the WHO has climbed from 6 to 11, the price was certain to rise. But the jump is largely attributable to the high price of new vaccines—particularly those for rotavirus and pneumococcal disease, which together make up more than 70 percent of the price tag—resulting in a cost structure that is simply not sustainable for many countries. GAVI has committed almost $8 billion dollars to introduce new vaccines and strengthen existing immunization systems in the developing world, and as the largest purchaser of vaccines for poorer countries, the organization can negotiate more favorable terms for countries it covers. But once a country’s annual per capita income rises above $1,550, that country begins to “graduate” and move out from under the GAVI umbrella, into terrain where they’ll be forced to pay incrementally more in the years that follow until they pay the full GAVI price, ultimately followed by paying full market prices. By 2016, for instance, some 17 countries will have lost GAVI support, and more countries are starting the “graduation” process each year. No longer eligible for reduced prices, countries like Republic of Congo and Honduras may soon have to choose which of the vaccines they can afford and which ones their youngest citizens will have to go without. One step to correct this would be to loosen the stranglehold a handful of pharmaceutical companies have over the vaccine market, allowing greater competition that would drive down costs. Another step would be for GAVI to use its purchasing power to negotiate better prices, and, just as importantly, give organizations like MSF—organizations that are on the ground, ready to put in the effort to reach as many children as possible—access to vaccines at the prices they negotiate for governments. These various issues came together in South Sudan’s Yida refugee camp this past year. A huge influx of refugees from Sudan had overwhelmed the services on the ground and created a dismal environment in which disease could easily spread. MSF teams built medical facilities, saw tens of thousands of patients, and provided a wide range of urgently needed services. MSF also identified pneumococcal diseases as one of the main reasons children were dying in the camp and decided to pursue vaccinating children with the pneumococcal vaccine, as well as the pentavalent vaccine. These vaccines were not yet in South Sudan’s national immunization schedule, though, so MSF had to purchase them ourselves. But because GAVI does not make its prices available to nongovernmental organizations and humanitarian actors such as MSF, it took 11 months of lengthy negotiations and bureaucratic hurdles for MSF to secure access to the pneumococcal vaccine. MSF was finally able to obtain a limited number of doses of the vaccine directly from one of the manufacturers and began vaccinating children in the camp in July of this year. This experience points to the fact that a more workable solution is needed to allow humanitarian actors to respond quickly when needs arise. We know it is possible. Two years ago, for instance, the Serum Institute, together with partners like PATH launched a new meningitis vaccine called MenAfriVac, which protects children from a strain of meningitis (meningitis A) prominent in Africa’s so-called meningitis belt. Because the vaccine was developed to serve people, not profits, it is sold at an extremely reasonable price, just 50 cents per dose. MenAfriVac is an example of an innovation model implementing delinkage. Even better, MenAfriVac’s protection lasted longer than the vaccine that was previously in use. Its widespread rollout in the region has already led to a significant decrease in the number of meningitis outbreaks.
There is quite a fair amount of public investment going to vaccines, TB and hepatitis. If the research agendas of public institutions and universities could be positively influenced to better respond to the needs of patients, many things could change. We need your voice and your activism in actively calling on governments, donors and universities to focus research on real public health needs and gaps, and to include access and affordability strategies from the beginning of the R&D process, with for example, the implementation of delinkage and the use of humanitarian and equitable pricing and technology licensing strategies.
Indeed we've won a few battles in the fight for access to medicines: we’ve secured price reductions of first line ARVs, whose prices have dropped by 99% in the last 10 years, and increased treatment access scale up to nearly 10 million people today. The Doha Declaration in 2001 was a turning point in the fight for access when it placed public health ahead of commercial interests. From 2002 to 2010, official development assistance committed to health more than quadrupled, from 4.4 billion USD in 2002 to 18.4 billion USD in 2010, with the US government contributing a third of it.
We all remember in 2001 the victory by the South African government over 39 drug companies attempting to block legislation that made medicine more affordable for the country. More recently, the Indian Supreme Court denial of a patent to Novartis drug Gleevec marked their affirmation to protect the availability of cheap generic drugs for poor patients and the right that governments have to fight patent evergreening.
These were important milestones, but we haven’t won the war. We've made tremendous progress in understanding how the current system functions and where it fails, but we haven't fixed it. And as the current system evolves we are faced with emerging challenges and persistent questions.
How can we align economic interests and public health imperatives, especially for underserved populations? As my colleague Judit Rius Sanjuan presented this morning, we are closely following the CEWG process, which we think could offer some opportunities of reforming the current model of innovation and research
How can we allow science to work in a truly open, collaborative fashion, serving public health needs? How can we make new science more affordable? We know how generic competition worked to lower prices for HIV drugs, but can those lessons be applied to other diseases and new technologies, and particularly biological products?
UAEM can be instrumental in proposing and forcing solutions to these problems, as we’ve seen with UAEM’s stavudine campaign and continued commitment to holding universities accountable through initiatives like the UAEM report cards and numerous statements of principle sign-ons. You are well equipped to reach out to researchers, academics and other stakeholders who are key in this dialogue. You will be the practitioner, witnessing, diagnosing and treating the patient's plight, you will be the brain that science needs to evolve, you will be the lawyer who can help protect the rights of the patients and you will be and ARE the activist who can resist and refuse the current status quo. I would like to emphasize this last point. Thanks to your presence on university campuses, you are uniquely positioned to demand changes in the approaches of universities for specific compounds. We, at MSF rely on your activism. We rely on you to map public and university funding spent on key promising drug candidates in TB, hep C, HIV and vaccines and pressure universities to only sign a license agreement for these drugs when the agreement fully promotes open innovation and access. We also need you to help us push universities/researchers/academics to promote open, collaborative models of research & development, maximum transparency of R&D data, innovative ways to manage IP (e.g. pooling) and delinkage.
In MSF, people come and go. Some do one field assignment. Others – like me - are involved with the organization for much longer. Regardless how long we've worked for this organization, our experience stays in us forever and changes our vision of humanity. UAEM and MSF have this passion in common. Whatever you decide to do in the future, I am convinced that your involvement at UAEM will always remain a source of inspiration and pride, and that the aspiration to address the patients’ needs will remain central to your work.
Thank you for your attention. It has been an honor to be able to speak to you today.
Médecins Sans Frontières/International Union Against Tuberculosis and Lung Disease. DR-TB Drugs Under the Microscope, 3rd edition. Geneva: Médecins Sans Frontières/International Union Against Tuberculosis and Lung Disease; 2013 Oct.
MSF obtained estimates.
Introduction
My life with MSF started in the early nineties in Thailand. MSF was providing medical care in Burmese refugee camps along the Thai-Burmese border. Malaria was the main cause of morbidity and mortality among this population. It was resistant to most of the available drugs to the point that young children and pregnant woman had become untreatable. The MSF team was frustrated with this situation—they documented the resistances and looked for alternatives without finding a solution.Then some patients came to us with a Chinese drug smuggled into Thailand through Burma. This drug was based on artemisinin derivatives. This family of new drugs was poorly known outside China. In fact, during the Vietnam War, for both the US and the Vietnamese forces, malaria was a real burden, keeping large numbers of soldiers out of combat. The Vietnamese asked their Chinese ally to provide them with better treatments. Chinese academics first screened the plants used in their traditional medicine. This is how the artemisinin derivatives were re-discovered. Meanwhile, Western laboratories, especially the US military research institutions, identified several new molecules, among them mefloquine and halofantrine.
The Vietnam War was over before this new generation of drugs had any impact on the treatment of military forces. Nonetheless, the two US drugs were developed and we used them in our programs. Later on, the parasite, Plasmodium falciparum, rapidly developed resistance against mefloquine and halofantrine. This is where the story starts for us and when we turned to the Chinese version.
With the Chinese drugs, three main difficulties immediately emerged: first of all, these drugs had not been studied according to international scientific standards; secondly, they were not economically attractive for big PHARMA to invest in because malaria was a disease of the poor, and also because the initial products were not patentable by Western pharmaceutical companies under the Chinese law at the time. And thirdly, politically, even psychologically, it was difficult for Western institutions to recognize that Chinese medical institutions had won the medical battle and found an alternative to outdated malaria drugs.
The first step out of this deadlock was to carry out clinical trials using alternative treatments. This was absolutely necessary if we were to have a chance at developing an effective treatment for one of the world’s most common and deadly diseases before it became completely untreatable. Looking at epidemiological data, it was obvious that there was a crucial need for a new treatment for malaria in other parts of the world, too. At the end of the nineties, the situation was rapidly worsening in sub-Saharan Africa. So between 1996 and 2004, MSF enrolled 12,000 patients in 43 clinical trials in 18 countries. Our efficacy studies were conducted according to scientific standards and published in peer-reviewed journals, and provided evidence on the efficacy of most drug regimens containing artesunate. We also supported the founding of the Drugs for Neglected Diseases initiative (DNDi) in 2003, whose mission was to fast track the development of drugs for neglected diseases. In addition to assisting in the development of new regimens to treat sleeping sickness, visceral leishmaniasis, and Chagas disease, DNDi helped create ASAQ and ASMQ, two single-dose combination treatments for malaria that include artemisinin and that are in widespread use in Africa and Asia today.
Artemisinin-based treatments are now part of WHO recommended protocols and have become a reference therapeutic option for malaria everywhere in the world. But as we speak, our teams in Cambodia are seeing the first signs of resistance to this treatment. We are already certain that this resistance will continue to spread and we can even anticipate which route it’ll take. But there is no single alternative treatment in sight.
The reason I shared this story on malaria with you is that in my view it is emblematic of a number of challenges that we still face today in our medical interventions: first, the lack of needs-driven innovation and research, especially when patients are poor and neglected; second, the persistence of numerous barriers (mostly economic) to access; and third, the need to fundamentally change the system in the long-run to promote innovation and access.
In my talk today, I will focus primarily on TB, vaccines, and hepatitis C, three areas where innovation and access gaps in the response from the global health system are currently causing tremendous suffering among the patients we serve. These are going to be three key priorities for MSF in the coming years. The fight for access to medicines by MSF always originates from a very concrete and practical problem faced by patients. It is not a moral or intellectual campaign that we embrace simply because we think it is the right cause.
Lack of a Needs-Driven R&D System
Let’s start with the lack of a needs-driven R&D system: Most of the time, our difficulty to treat patients will come from a lack of appropriate treatment, vaccine, or diagnostic tool, and a lack of attention to groups that are particularly vulnerable. We all know the old estimate that less than 10% of research is committed to diseases and conditions that account for 90% of the global disease burden. Last year, we organized an innovation conference in New York with DNDi and Mount Sinai. We decided to look at the numbers again. The resulting study, which was published in the Lancet just a few weeks ago, shows that, despite some progress, between 2000 and 2011 only 3.4% of all new drug approvals were indicated for neglected diseases. Meanwhile the global burden of disease is estimated at 11%. It is worth pointing out that of these products, only 4 (or 1%) were truly new chemical entities, 3 of which were approved for malaria, and no new drugs for TB or neglected tropical diseases were approved over the decade. Most newly developed therapeutic products were repurposed versions of existing drugs. Our conclusion is simple: the system is broken and a "fatal imbalance" remains in R&D for many neglected patients.Take vaccines and TB for example: Vaccines have long been part of the services MSF offers, particularly in countries where vaccine coverage is low. As part of basic health care programs in these places, teams provide children with a slate of vaccines currently recommended by the World Health Organization: DTP (diphtheria, tetanus, pertussis), hepatitis B, Haemophilius influenzae type b (Hib), BCG (against tuberculosis), measles, polio, and, increasingly, pneumococcal conjugate vaccine.
According to WHO estimates, globally in 2012 more than 22 million children did not benefit from basic vaccination. One big barrier to better coverage is that in rural areas of resource-limited settings, parents have to travel far to get their children vaccinated, simply because in many cases, it’s not possible to bring the vaccines to them. At present, these vaccines have storage and delivery requirements that can be very difficult to uphold in resource-limited settings. Imagine trying to reach children in a remote village in, say, India, or Chad, or Central African Republic. The roads are bad and prone to flooding during the rainy season. The vaccines themselves need to be kept at a certain temperature—between 2° to 8° Celsius, or roughly 35° to 46° Fahrenheit—lest they become ineffective, but there’s no electricity along much of the route. And most vaccines have to be administered with needles by trained health professionals, who are often in short supply in countries lagging in terms of development or afflicted by years of conflict. MSF teams must contend with all of these challenges, from the logistics of the cold chains, to the provision of trained medical staff. There is a profound need for vaccines that are better-adapted to the settings in which they will be used. If they could withstand higher temperatures, if they could be delivered through a mist or a patch, or if the course could be completed with fewer doses, it would be far more feasible to get the vaccines and health workers to the most decentralized health facilities, closer to where most people live. Rather than sticking with a one-size-fits-all approach—that is usually based on US and European models—vaccines R&D efforts should be much more global, responding to the needs of all children and the health systems that try to reach them.
Moving on to tuberculosis: After more than 4 decades of inaction, as we speak, two new promising drugs for TB are coming to market, but very little effort has been made by their producers to assess the drugs’ efficacy in new combination treatments or in sharing clinical safety data that would allow others to do this work. Effective TB treatment requires a robust combination of different classes of drugs to prevent development of resistance to individual drugs. So, even though the advent of new promising TB drugs is a milestone for us and for the 30,000 TB patients we treat in a year, these new drugs won’t actually do much to curb the drug-resistant TB epidemic until we know how to combine them and what kind of regimen is appropriate for treatment, and results from the necessary clinical trials are still many years away. Furthermore, producers have little incentive to make these new drugs affordable for all those who, from South Africa to Uzbekistan, are currently dying from drug-resistant TB and who desperately need access to new and better treatment regimens. So, as with malaria 20 years ago, big pharma lacks any incentive to perform the research necessary to meet patient needs. Companies are incentivized to work in silos to bring individual drugs to market, rather than to perform the collaborative research needed to develop and bring new regimens to market. We face a future where eventually treatment providers and NGOs will have to step up and conduct clinical trials themselves as we had to in malaria.
And that is why we need a fundamental change in the way in which medical research and development is conducted, and this is what MSF, UAEM, and many others are trying to enact through the WHO. As part of the implementation of the 2008 WHO Global Strategy and Plan of Action on Public Health, Innovation, and Intellectual Property and some of the recommendations included in the 2012 report by the Consultative Expert Working Group on Research and Development: Financing and Coordination (the CEWG Report), MSF has submitted two proposals for demonstration projects on innovative incentive and funding mechanisms for R&D: one for open-source, multiplex, affordable fever diagnostics and another for a new framework for TB regimen development.
Allow me to briefly explain our TB proposal, called the 3P Project (Push, Pull, Pool). We are proposing the creation of an open, collaborative framework for TB drug regimen development based on the sharing of data, the pooling of intellectual property, and the creation of incentives in the form of prizes and grants. Our proposal encourages multiple actors to enter the R&D process in order to accelerate development timelines, improve R&D effectiveness, and provide more equitable access to better medicines. The proposal de-links the costs of research and development from the prices of the end products created (known as “delinkage”) and embraces open, collaborative innovation models.
Your help in supporting our efforts to explain the CEWG process and the MSF proposals (as well as other proposals) to academics, researchers, universities, and many others would be extremely valuable. We need to broaden the discussion beyond the corridors of Geneva.
MSF has also launched an online TB manifesto advocating for better treatments that I encourage you all to sign and share broadly.
Access Barriers
My second point relates to the newer access barriers we face. We know too well that access to medicine is not just a matter of finding the right drug or adapting tools to the contexts in which they’ll be deployed, it is about making them accessible and affordable to those who need them most. The countries where MSF intervenes today present a very different epidemiologic profile than in the past decade, from Pakistan to DRC, and from Syria to Kenya. We no longer have to deal only with a few rare tropical diseases, or only the poorest countries. Many of the most difficult access challenges are going to be in developing countries that are classified as middle-income economies. And apart from having to ensure access to diagnostics and treatments for the big three - HIV, TB and malaria, as in the US and Europe, non-communicable diseases, or NCDs, such as cancer, hypertension, diabetes, and cardiovascular diseases are more and more common and widespread among the populations we are serving. Existing alternatives for us and for the countries where we work are limited to treatments and tools that are most of the time outrageously expensive. MSF is joining late in the game of NCDs. Although we are of course treating patients affected with some NCDs, we have not yet fully adapted our emergency set up to these pathologies, nor have we properly documented the state of affairs, which would allow us to engage in a more aggressive campaign. This is a work in process.In other areas however, challenges of access and affordability are quite obvious. After more than 40 years with no new drugs, the newly FDA-approved TB treatment bedaquiline will most likely cost around 900 USD for a 6-month treatment course in least developed countries, and 3,000 USD for a six-month course in middle income countries. That’s for just one drug, but several drugs are needed for effective TB treatment. This means that new treatment combinations will still cost in excess of several thousand dollars per patient even in the poorest countries, as they do today , and potentially much more in countries classified as ‘middle-income,’ of which many have a high burden of TB, such as India, Ukraine and Uzbekistan.
At least 185 million people worldwide have been infected with hepatitis C virus (HCV), but access to treatment is and will continue to be a huge challenge. The current treatment peginterferon is expensive and not ideal in terms of medical outcomes, but some very promising new hepatitis C oral treatments are coming out of the pipeline very soon. The problem is that they are going to be completely unaffordable for many. Let me give you one example: sofubsuvir from Gilead is going to receive FDA and European regulatory approval very soon. According to the latest estimates, it could be priced in the range of 50-80,000 USD for a treatment course in wealthy countries. And there is yet no public indication on what Gilead will charge for its use in developing countries. We know that competition, if allowed, could be a game changer because the cost of manufacturing has been estimated to be around 200 USD per treatment.
Bringing prices of vaccines down is another crucial priority. In 2001, the cost to provide a child with basic vaccinations was $1.38. Today, it’s $38.80, an increase of more than 2,700 percent. Given the fact that the number of basic antigens recommended by the WHO has climbed from 6 to 11, the price was certain to rise. But the jump is largely attributable to the high price of new vaccines—particularly those for rotavirus and pneumococcal disease, which together make up more than 70 percent of the price tag—resulting in a cost structure that is simply not sustainable for many countries. GAVI has committed almost $8 billion dollars to introduce new vaccines and strengthen existing immunization systems in the developing world, and as the largest purchaser of vaccines for poorer countries, the organization can negotiate more favorable terms for countries it covers. But once a country’s annual per capita income rises above $1,550, that country begins to “graduate” and move out from under the GAVI umbrella, into terrain where they’ll be forced to pay incrementally more in the years that follow until they pay the full GAVI price, ultimately followed by paying full market prices. By 2016, for instance, some 17 countries will have lost GAVI support, and more countries are starting the “graduation” process each year. No longer eligible for reduced prices, countries like Republic of Congo and Honduras may soon have to choose which of the vaccines they can afford and which ones their youngest citizens will have to go without. One step to correct this would be to loosen the stranglehold a handful of pharmaceutical companies have over the vaccine market, allowing greater competition that would drive down costs. Another step would be for GAVI to use its purchasing power to negotiate better prices, and, just as importantly, give organizations like MSF—organizations that are on the ground, ready to put in the effort to reach as many children as possible—access to vaccines at the prices they negotiate for governments. These various issues came together in South Sudan’s Yida refugee camp this past year. A huge influx of refugees from Sudan had overwhelmed the services on the ground and created a dismal environment in which disease could easily spread. MSF teams built medical facilities, saw tens of thousands of patients, and provided a wide range of urgently needed services. MSF also identified pneumococcal diseases as one of the main reasons children were dying in the camp and decided to pursue vaccinating children with the pneumococcal vaccine, as well as the pentavalent vaccine. These vaccines were not yet in South Sudan’s national immunization schedule, though, so MSF had to purchase them ourselves. But because GAVI does not make its prices available to nongovernmental organizations and humanitarian actors such as MSF, it took 11 months of lengthy negotiations and bureaucratic hurdles for MSF to secure access to the pneumococcal vaccine. MSF was finally able to obtain a limited number of doses of the vaccine directly from one of the manufacturers and began vaccinating children in the camp in July of this year. This experience points to the fact that a more workable solution is needed to allow humanitarian actors to respond quickly when needs arise. We know it is possible. Two years ago, for instance, the Serum Institute, together with partners like PATH launched a new meningitis vaccine called MenAfriVac, which protects children from a strain of meningitis (meningitis A) prominent in Africa’s so-called meningitis belt. Because the vaccine was developed to serve people, not profits, it is sold at an extremely reasonable price, just 50 cents per dose. MenAfriVac is an example of an innovation model implementing delinkage. Even better, MenAfriVac’s protection lasted longer than the vaccine that was previously in use. Its widespread rollout in the region has already led to a significant decrease in the number of meningitis outbreaks.
There is quite a fair amount of public investment going to vaccines, TB and hepatitis. If the research agendas of public institutions and universities could be positively influenced to better respond to the needs of patients, many things could change. We need your voice and your activism in actively calling on governments, donors and universities to focus research on real public health needs and gaps, and to include access and affordability strategies from the beginning of the R&D process, with for example, the implementation of delinkage and the use of humanitarian and equitable pricing and technology licensing strategies.
The need for a new system
Let me now raise my third and last point: the malaria story illustrates how history repeats itself over and over again. Most of the time with the same cynicism and equally tragic consequences for the patients. Today, with the emerging resistance to artemisinin, we are back to square one. As the film’s narrator pointed out in the recently released documentary, Fire in the Blood: the drug companies have lost some of the battles for access, but they certainly haven’t lost the war.Indeed we've won a few battles in the fight for access to medicines: we’ve secured price reductions of first line ARVs, whose prices have dropped by 99% in the last 10 years, and increased treatment access scale up to nearly 10 million people today. The Doha Declaration in 2001 was a turning point in the fight for access when it placed public health ahead of commercial interests. From 2002 to 2010, official development assistance committed to health more than quadrupled, from 4.4 billion USD in 2002 to 18.4 billion USD in 2010, with the US government contributing a third of it.
We all remember in 2001 the victory by the South African government over 39 drug companies attempting to block legislation that made medicine more affordable for the country. More recently, the Indian Supreme Court denial of a patent to Novartis drug Gleevec marked their affirmation to protect the availability of cheap generic drugs for poor patients and the right that governments have to fight patent evergreening.
These were important milestones, but we haven’t won the war. We've made tremendous progress in understanding how the current system functions and where it fails, but we haven't fixed it. And as the current system evolves we are faced with emerging challenges and persistent questions.
How can we align economic interests and public health imperatives, especially for underserved populations? As my colleague Judit Rius Sanjuan presented this morning, we are closely following the CEWG process, which we think could offer some opportunities of reforming the current model of innovation and research
How can we allow science to work in a truly open, collaborative fashion, serving public health needs? How can we make new science more affordable? We know how generic competition worked to lower prices for HIV drugs, but can those lessons be applied to other diseases and new technologies, and particularly biological products?
UAEM can be instrumental in proposing and forcing solutions to these problems, as we’ve seen with UAEM’s stavudine campaign and continued commitment to holding universities accountable through initiatives like the UAEM report cards and numerous statements of principle sign-ons. You are well equipped to reach out to researchers, academics and other stakeholders who are key in this dialogue. You will be the practitioner, witnessing, diagnosing and treating the patient's plight, you will be the brain that science needs to evolve, you will be the lawyer who can help protect the rights of the patients and you will be and ARE the activist who can resist and refuse the current status quo. I would like to emphasize this last point. Thanks to your presence on university campuses, you are uniquely positioned to demand changes in the approaches of universities for specific compounds. We, at MSF rely on your activism. We rely on you to map public and university funding spent on key promising drug candidates in TB, hep C, HIV and vaccines and pressure universities to only sign a license agreement for these drugs when the agreement fully promotes open innovation and access. We also need you to help us push universities/researchers/academics to promote open, collaborative models of research & development, maximum transparency of R&D data, innovative ways to manage IP (e.g. pooling) and delinkage.
Conclusion
We have many challenges ahead of us. You are the present and the future of the access to medicines movement. We hope that you will be instrumental in changing the paradigm toward a needs-based R&D system. One that will be patient-centered. One that does not require years before better medical products reach patients. A system where physicians don't have to beg for more effective or less toxic drugs and where we – non-profit organizations - are not forced to invest in our own clinical trial to offset the inertia of the global health community. A system where science can benefit all patients at a just price, and where the priority of saving as many lives as possible takes precedence over profit.In MSF, people come and go. Some do one field assignment. Others – like me - are involved with the organization for much longer. Regardless how long we've worked for this organization, our experience stays in us forever and changes our vision of humanity. UAEM and MSF have this passion in common. Whatever you decide to do in the future, I am convinced that your involvement at UAEM will always remain a source of inspiration and pride, and that the aspiration to address the patients’ needs will remain central to your work.
Thank you for your attention. It has been an honor to be able to speak to you today.
-Sophie Delaunay, MSF-USA Executive Director
Médecins Sans Frontières/International Union Against Tuberculosis and Lung Disease. DR-TB Drugs Under the Microscope, 3rd edition. Geneva: Médecins Sans Frontières/International Union Against Tuberculosis and Lung Disease; 2013 Oct.
MSF obtained estimates.
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Tuberculosis: The New Face of an Old Disease
March 17, 2014
AMSTERDAM/NEW YORK—Immediate action is needed from governments, pharmaceutical companies, and researchers to find new treatments for drug-resistant tuberculosis (DR-TB), and to prevent people from dying from one of the world’s most urgent global health threats, the international medical humanitarian organization Doctors Without Borders/Médecins Sans Frontières (MSF) said today in a report released in advance of World TB Day.
The MSF report draws on first-hand experience working on the frontline of the DR-TB crisis, and describes the critical need to diagnose and treat more people now, and to find new, much shorter, more tolerable, and more effective treatment combinations. Roughly eight million people worldwide fall ill with tuberculosis and 1.3 million people die from the infectious airborne disease every year.
TB is curable, but an inadequate global response has allowed drug-resistant TB to take hold. Around half a million new cases of multidrug-resistant TB (MDR-TB) occur every year, with cases reported in virtually all countries worldwide. Even harder-to-treat, extensively drug-resistant TB (XDR-TB) is documented in nearly 100 countries. While these deadlier drug-resistant strains are spreading from person to person, no effective treatment exists.
Although two new TB drugs were recently developed—the first in 40 years—doctors and patients are years away from the treatment revolution they need. To be effective, TB drugs have to be used in combination, and clinical trials combining the new drugs are not yet underway
The inadequate and costly treatment is severely undermining the global response. With only one in five people in need receiving DR-TB treatment today, the fatal, airborne disease is left to spread indiscriminately.
The severity of the crisis has led DR-TB patients and medical staff worldwide to come together to call for urgent improvements in "Test Me, Treat Me: A DR-TB Manifesto."
AMSTERDAM/NEW YORK—Immediate action is needed from governments, pharmaceutical companies, and researchers to find new treatments for drug-resistant tuberculosis (DR-TB), and to prevent people from dying from one of the world’s most urgent global health threats, the international medical humanitarian organization Doctors Without Borders/Médecins Sans Frontières (MSF) said today in a report released in advance of World TB Day.
The MSF report draws on first-hand experience working on the frontline of the DR-TB crisis, and describes the critical need to diagnose and treat more people now, and to find new, much shorter, more tolerable, and more effective treatment combinations. Roughly eight million people worldwide fall ill with tuberculosis and 1.3 million people die from the infectious airborne disease every year.
Read Drug-Resistant Tuberculosis: The New Face of an Old Disease
“The DR-TB crisis is everybody’s problem and demands an immediate international response,” said Dr Sidney Wong, MSF medical director. “Each year we are diagnosing more patients with DR-TB, but the current treatments aren’t good enough to make a dent in the epidemic. It doesn’t matter where you live; until new, short, and more effective treatment combinations are found, the odds of surviving this disease today are dismal.”TB is curable, but an inadequate global response has allowed drug-resistant TB to take hold. Around half a million new cases of multidrug-resistant TB (MDR-TB) occur every year, with cases reported in virtually all countries worldwide. Even harder-to-treat, extensively drug-resistant TB (XDR-TB) is documented in nearly 100 countries. While these deadlier drug-resistant strains are spreading from person to person, no effective treatment exists.
Video: A Breath of Hope for MDR-TB Patients
While the growing use of a new rapid diagnostic test for MDR-TB is helping to identify more and more patients, standard TB drugs won’t cure patients, and doctors must turn to long, complex, and expensive drugs that, at best, only cure half the patients on treatment. People face a two-year ordeal involving swallowing more than 10,000 pills and enduring eight months of daily injections. The treatments make many people seriously ill, with side effects ranging from nausea and body pain to permanent hearing loss and psychosis. The drugs alone cost health providers around US$4,000 per person per year, not including costs for long periods of care and the management of side effects.Although two new TB drugs were recently developed—the first in 40 years—doctors and patients are years away from the treatment revolution they need. To be effective, TB drugs have to be used in combination, and clinical trials combining the new drugs are not yet underway
The inadequate and costly treatment is severely undermining the global response. With only one in five people in need receiving DR-TB treatment today, the fatal, airborne disease is left to spread indiscriminately.
The severity of the crisis has led DR-TB patients and medical staff worldwide to come together to call for urgent improvements in "Test Me, Treat Me: A DR-TB Manifesto."
Video: New Drugs for Drug-Resistant TB Are a Lifeline
“Let us accept the fact we are faced with a TB epidemic,” said a mother of two from Swaziland, whose daughter died of DR-TB and whose son is now infected with the disease. “We need to get together and fight for our survival so that there can be a future for the next generation, because if we give up the fight now, the children are finished.”Tuberculosis: The New Face of an Old Disease
March 17, 2014
AMSTERDAM/NEW YORK—Immediate action is needed from governments, pharmaceutical companies, and researchers to find new treatments for drug-resistant tuberculosis (DR-TB), and to prevent people from dying from one of the world’s most urgent global health threats, the international medical humanitarian organization Doctors Without Borders/Médecins Sans Frontières (MSF) said today in a report released in advance of World TB Day.
The MSF report draws on first-hand experience working on the frontline of the DR-TB crisis, and describes the critical need to diagnose and treat more people now, and to find new, much shorter, more tolerable, and more effective treatment combinations. Roughly eight million people worldwide fall ill with tuberculosis and 1.3 million people die from the infectious airborne disease every year.
TB is curable, but an inadequate global response has allowed drug-resistant TB to take hold. Around half a million new cases of multidrug-resistant TB (MDR-TB) occur every year, with cases reported in virtually all countries worldwide. Even harder-to-treat, extensively drug-resistant TB (XDR-TB) is documented in nearly 100 countries. While these deadlier drug-resistant strains are spreading from person to person, no effective treatment exists.
Although two new TB drugs were recently developed—the first in 40 years—doctors and patients are years away from the treatment revolution they need. To be effective, TB drugs have to be used in combination, and clinical trials combining the new drugs are not yet underway
The inadequate and costly treatment is severely undermining the global response. With only one in five people in need receiving DR-TB treatment today, the fatal, airborne disease is left to spread indiscriminately.
The severity of the crisis has led DR-TB patients and medical staff worldwide to come together to call for urgent improvements in "Test Me, Treat Me: A DR-TB Manifesto."
A common problem in New York is a lack of interest or inclination on the relationship of the immune system
to various diseases.
Before you even think about Winthrop support Amtrak and to to Boston after you have read the work of
Dr. Denise L Faustman, see eg faustmanlab.org and pubmed.org faustman dl
If you are Italian, love Italy and have MS or some other autoimmune disease read pubmed.org ristori bcg and travel to Italy or simply obtain your BCG and shoot it . It is safe, effective and inexpensive.
The Healthcare Motto of the US is simply that of the great Dr. Hiram Maxim.
Lead, the safest, cheapest and guaranteed effective treatment of all. It has been vetted in countless wars over many years and every country in the world owes a debt of gratitude to Dr. Hiram Maxim who has cured every condition known to man inexpensively .
AMSTERDAM/NEW YORK—Immediate action is needed from governments, pharmaceutical companies, and researchers to find new treatments for drug-resistant tuberculosis (DR-TB), and to prevent people from dying from one of the world’s most urgent global health threats, the international medical humanitarian organization Doctors Without Borders/Médecins Sans Frontières (MSF) said today in a report released in advance of World TB Day.
The MSF report draws on first-hand experience working on the frontline of the DR-TB crisis, and describes the critical need to diagnose and treat more people now, and to find new, much shorter, more tolerable, and more effective treatment combinations. Roughly eight million people worldwide fall ill with tuberculosis and 1.3 million people die from the infectious airborne disease every year.
Read Drug-Resistant Tuberculosis: The New Face of an Old Disease
“The DR-TB crisis is everybody’s problem and demands an immediate international response,” said Dr Sidney Wong, MSF medical director. “Each year we are diagnosing more patients with DR-TB, but the current treatments aren’t good enough to make a dent in the epidemic. It doesn’t matter where you live; until new, short, and more effective treatment combinations are found, the odds of surviving this disease today are dismal.”TB is curable, but an inadequate global response has allowed drug-resistant TB to take hold. Around half a million new cases of multidrug-resistant TB (MDR-TB) occur every year, with cases reported in virtually all countries worldwide. Even harder-to-treat, extensively drug-resistant TB (XDR-TB) is documented in nearly 100 countries. While these deadlier drug-resistant strains are spreading from person to person, no effective treatment exists.
Video: A Breath of Hope for MDR-TB Patients
While the growing use of a new rapid diagnostic test for MDR-TB is helping to identify more and more patients, standard TB drugs won’t cure patients, and doctors must turn to long, complex, and expensive drugs that, at best, only cure half the patients on treatment. People face a two-year ordeal involving swallowing more than 10,000 pills and enduring eight months of daily injections. The treatments make many people seriously ill, with side effects ranging from nausea and body pain to permanent hearing loss and psychosis. The drugs alone cost health providers around US$4,000 per person per year, not including costs for long periods of care and the management of side effects.Although two new TB drugs were recently developed—the first in 40 years—doctors and patients are years away from the treatment revolution they need. To be effective, TB drugs have to be used in combination, and clinical trials combining the new drugs are not yet underway
The inadequate and costly treatment is severely undermining the global response. With only one in five people in need receiving DR-TB treatment today, the fatal, airborne disease is left to spread indiscriminately.
The severity of the crisis has led DR-TB patients and medical staff worldwide to come together to call for urgent improvements in "Test Me, Treat Me: A DR-TB Manifesto."
Video: New Drugs for Drug-Resistant TB Are a Lifeline
“Let us accept the fact we are faced with a TB epidemic,” said a mother of two from Swaziland, whose daughter died of DR-TB and whose son is now infected with the disease. “We need to get together and fight for our survival so that there can be a future for the next generation, because if we give up the fight now, the children are finished.”A common problem in New York is a lack of interest or inclination on the relationship of the immune system
to various diseases.
Before you even think about Winthrop support Amtrak and to to Boston after you have read the work of
Dr. Denise L Faustman, see eg faustmanlab.org and pubmed.org faustman dl
If you are Italian, love Italy and have MS or some other autoimmune disease read pubmed.org ristori bcg and travel to Italy or simply obtain your BCG and shoot it . It is safe, effective and inexpensive.
The Healthcare Motto of the US is simply that of the great Dr. Hiram Maxim.
Lead, the safest, cheapest and guaranteed effective treatment of all. It has been vetted in countless wars over many years and every country in the world owes a debt of gratitude to Dr. Hiram Maxim who has cured every condition known to man inexpensively .
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Diabetes, a public health threat of epidemic proportions, does not
discriminate. It affects every culture, race, age group and gender.
Type 2 diabetes is closely linked to the rapid increase in obesity. It
accounts for over 90% of all people with diabetes. Type 1 diabetes
affects less than 10% of people with diabetes but typically starts in
children and always requires insulin replacement. It is caused by the
body’s immune responses wiping out cells that make insulin.
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