Thursday, July 24, 2014

Death by CARDIOLOGIST and he

Torture by NEUROLOGISTS IF  YOU HAVE MULTIPLE SCLEROSIS.
BETTER THE BOSTON AND ROME ALTERNATIVES. 

1.  IF YOU HAVE SEVERE PSORIASIS you are at increased risk of cardiovascular complications.
2. According to Dr. Denise L Faustman, see faustmanlab.org, it is best to increase the level of TNF alpha rather than decrease same.
3 I am a (human) MONKEY WITH TYPE 1 DIABETES AND PLAQUE PSORIASIS.
4. I have found that plaque psoriasis responds to BCG vaccinations.
5. Rather than pay your cardiologist to kill you I will pay you to (kill me) AND demonstrate on You Tube that injections of BCG treats plaque psoriasis.

You will find ample scientific publications to support the use of BCG .See eg pubmed.org



Increased serum leptin and resistin levels and increased carotid intima-media wall thickness in patients with psoriasis: Is psoriasis associated with atherosclerosis?
Robati RM, Partovi-Kia M, Haghighatkhah HR, Younespour S, Abdollahimajd F.
J Am Acad Dermatol. 2014 Jul 16. pii: S0190-9622(14)01567-9. doi: 10.1016/j.jaad.2014.06.006. [Epub ahead of print]
PMID:
25043845
[PubMed - as supplied by publisher]
Related citations
2.
Influence of IL-6 gene polymorphisms in epicardial adipose tissue and coronary artery calcification in psoriasis patients.
Torres T, Bettencourt N, Ferreira J, Carvalho C, Mendonça D, Pinho-Costa P, Vasconcelos C, Selores M, Silva B.
Br J Dermatol. 2014 Jul 17. doi: 10.1111/bjd.13271. [Epub ahead of print]
PMID:
25040543
[PubMed - as supplied by publisher]
Related citations
3.
Change in arterial intima-media thickness: Is it an appropriate surrogate for cardiovascular risk in psoriasis patients treated with tumor necrosis factor-alpha inhibitors?
Dominguez AR, Condie D.
J Am Acad Dermatol. 2014 Aug;71(2):388-9. doi: 10.1016/j.jaad.2014.02.048. No abstract available.
PMID:
25037785
[PubMed - in process]
Related citations

PUTTING THE US HEALTHCARE SYSTEM TO SHAME.  SUPPORT ITALY.
 
Effects of Bacille Calmette-Guerin after the first demyelinating event in the CNS.
Ristori G, Romano S, Cannoni S, Visconti A, Tinelli E, Mendozzi L, Cecconi P, Lanzillo R, Quarantelli M, Buttinelli C, Gasperini C, Frontoni M, Coarelli G, Caputo D, Bresciamorra V, Vanacore N, Pozzilli C, Salvetti M.
Neurology. 2014 Jan 7;82(1):41-8. doi: 10.1212/01.wnl.0000438216.93319.ab. Epub 2013 Dec 4.
PMID:
24306002
[PubMed - indexed for MEDLINE]
Related citations


 
TNF Receptor 2 and Disease: Autoimmunity and Regenerative Medicine.
Faustman DL, Davis M.
Front Immunol. 2013 Dec 23;4:478. doi: 10.3389/fimmu.2013.00478.
PMID:
24391650
[PubMed]
Free PMC Article
Related citations

 
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Cell Mol Life Sci. 2005 Aug;62(16):1850-62.

The therapeutic potential of tumor necrosis factor for autoimmune disease: a mechanistically based hypothesis.

Kodama S1, Davis M, Faustman DL.

Author information

Abstract

Excess levels of tumor necrosis factor-alpha (TNF-alpha) have been associated with certain autoimmune diseases. Under the rationale that elevated TNF-alpha levels are deleterious, several anti-TNF-alpha therapies are now available to block the action of TNF-alpha in patients with autoimmune diseases with a chronic inflammatory component to the destructive process. TNF-alpha antagonists have provided clinical benefit to many patients, but their use also is accompanied by new or aggravated forms of autoimmunity. Here we propose a mechanistically based hypothesis for the adverse events observed with TNF-alpha antagonists, and argue for the opposite therapeutic strategy: to boost or restore TNF-alpha activity as a treatment for some forms of autoimmunity. Activation defects in the transcription factor nuclear factor kappaB leave autoreactive T cells sensitive to TNF-alpha-induced apoptosis. Treatment with TNF-alpha, by destroying autoreactive T cells, appears to be a highly targeted strategy to interrupt the pathogenesis of type 1 diabetes, lupus and certain forms of autoimmunity.
PMID:
15968469
[PubMed - indexed for MEDLINE]
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